The onset of Kidney Disease is insidious, and many patients do not find it until CKD 5 stage. When nephropathy is first discovered, which patients are prone to End Stage Renal Disease. Following this article to get answer, or you can consult ONLINE DOCTOR directly in free.
Henoch-Schonlein purpura nephritis mostly occurs in young people, and most of them are easy to control. Most patients after treatment, urinary protein, urinary occult blood will turn negative.
Some patients, as well as their children's parents, were completely reassured after their purpura kidney improved and stopped taking medicine, and they did not go for further review.
However, Purpura Nephropathy tends to recur, especially in male patients and adult patients, the prognosis is not very good. Henoch-Schonlein purpura kidney does not necessarily have spots on the skin when it develops, or there may be no obvious physical cues. It may not be until renal failure or uremia that the condition deteriorates.
Acute nephritis can almost be cured after treatment. But the patient suffers from acute nephritis for a reason, urinary tract infection, poor immunity and other factors, but also related to life habits. The acute nephritis was cured this time, but the risk of relapse was still higher than that of other people.
After cure, patients with acute nephritis still need regular blood pressure measurement, urinary routine examination and renal function.
Hematuria is the main manifestation of chronic nephritis patients, usually urinary protein is not too high, many patients rely on "naked eye hematuria" and found themselves suffering from nephritis.
When chronic glomerulonephritis recurs again, there is not necessarily naked eye hematuria, if there is no edema, patients do not have the habit of taking blood pressure, it is difficult to find the progress of the disease. Perhaps the patient will not feel the symptoms until uremia.
Hypertension first damages the renal interstitium and renal tubules, rather than the glomeruli. Therefore, in the early stage of hypertensive renal damage, the urine protein is normal, and there will be no edema, urine foam and so on.
When the tubulointerstitial injury becomes serious, the glomerulus will also be involved, resulting in a comprehensive kidney atrophy. Perhaps when the renal failure caused by hypertension progresses to the end stage (uremia), the body will inform you that you have kidney disease.
Like hypertension, renal damage in diabetes mellitus begins with tubulointerstitial damage. When diabetic nephropathy develops to phase 3. Renal function decreases with the increase of urinary protein.
Diabetic nephropathy is different from primary nephropathy. Primary nephropathy usually begins with proteinuria, and renal failure occurs many years later. Diabetic nephropathy, clinical proteinuria and renal failure occur simultaneously.
So diabetic patients need to regularly check the urinary microalbumin, and check the kidney damage when the urinary protein is not obvious.
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